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 Your Results - you searched for the keyword Neuroendocrinology 7 Results    

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1. Bergmann, U. (2011, August). Acute, chronic and complex PTSD: Exploring their neuroendocrinology and relationship to medically unexplained symptoms. Presentation at the annual meeting of the EMDR International Association, Orange County, CA.

Language: English

Format: Conference

Abstract:
What exactly happens on a neuroendocrine level in acute, chronic and complex PTSD? Is there a relationship between this unusual neuroendocrine profile and a number of medical disorders of unknown origin which, often, do not respond to traditional symptomatic medical treatment? This seminar will review and examine the results of extensive neuroendocrine research relative to these trauma-induced disorders. The data from these research bases will be integrated with neuroendocrine research findings regarding autoimmune compromises associated with chronic trauma. Anomalous conditions, such as Fibromyalgia, Chronic Fatigue Syndrome, Reflex Sympathetic Dystrophy (RSD), Systemic Lupus Erythematosis, Hashimoto’s Thyroiditis, Sjogren’s Syndrome, and Rheumatoid Arthritis will be examined. This presentation will illustrate the neuroendocrine and causal relationship between the various forms of PTSD and these medical disorders. Accordingly, the implications for treatment will be examined.

Keywords: Acute PTSD  Chronic PTSD  Complex Posttraumatic Stress Disorder  Complex PTSD  C-PTSD  Medically-Unexplained Sysmptoms  Neuroendocrinology  

Accuracy Verified: Yes

2. Bergmann, U. (2012, October). Acute, chronic and complex PTSD: Exploring their neuroendocrinology and relationship to medically unexplained symptoms. Presentation at the annual meeting of the EMDR International Association, Arlington, VA.

Language: English

Format: Conference

Abstract:
What exactly happens on a neuroendocrine level in acute, chronic and complex PTSD? Is there a relationship between this unusual neuroendocrine profile and a number of medical disorders of unknown origin which, often, do not respond to traditional symptomatic medical treatment? This seminar will review and examine the results of extensive neuroendocrine research relative to these trauma-induced disorders. The data from these research bases will be integrated with neuroendocrine research findings regarding autoimmune compromises associated with chronic trauma. Anomalous conditions, such as Fibromyalgia, Chronic Fatigue Syndrome, Reflex Sympathetic Dystrophy (RSD), Systemic Lupus Erythematosis, Hashimoto’s Thyroiditis, Sjogren’s Syndrome, and Rheumatoid Arthritis will be examined. This presentation will illustrate the neuroendocrine and causal relationship between the various forms of PTSD and these medical disorders. Accordingly, the implications for EMDR treatment will be examined, as well as referrals for medical treatment.

Keywords: Acute PTSD  Chronic PTSD  Complex PTSD  Medically Unexplained Symptoms  Neuroendocrinology  

Accuracy Verified: Yes

3. Solomon, E. P., & Heide, K. M. (2005, January). The biology of trauma:  Implications for treatment. Journal of Interpersonal Violence, 20(1), 51-60. doi:10.1177/0886260504268119.

Language: English

Format: Journal

Abstract:
During the past 20 years, the development of brain imaging techniques and new biochemical approaches has led to increased understanding of the biological effects of psychological trauma. New hypotheses have been generated about brain development and the roots of antisocial behavior. We now understand that psychological trauma disrupts homeostasis and can cause both short and long-term effects on many organs and systems of the body. Our expanding knowledge of the effects of trauma on the body has inspired new approaches to treating trauma survivors. Biologically informed therapy addresses the physiological effects of trauma, as well as cognitive distortions and maladaptive behaviors. The authors suggest that the most effective therapeutic innovation during the past 20 years for treating trauma survivors has been Eye Movement Desensitization and Reprocessing (EMDR), a therapeutic approach that focuses on resolving trauma using a combination of top-down (cognitive) and bottom-up (affect/body) processing. [Author Abstract]

Keywords: Adolescents  Child Abuse  Children  Criminal Behavior  Forensic Evaluation  Literature Review  Neglect  Neuroendocrinology  Neurophysiology  Posttraumatic Stress Disorder  PTSD  Survivors  

Accuracy Verified: Yes

4. Heide, K. M., & Solomon, E. P. (2006, May-June). Biology, childhood trauma, and murder: Rethinking justice. International Journal of Law and Psychiatry, 29(3), 220-233. doi:10.1016/j.ijlp.2005.10.001.

Language: English

Format: Journal

Abstract:
This article reviews recent findings in the developmental neurophysiology of children subjected to psychological trauma. Studies link extreme neglect and abuse with long-term changes in the nervous and endocrine systems. A growing body of research literature indicates that individuals with severe trauma histories are at higher risk of behaving violently than those without such histories. This article links these two research areas by discussing how severe and protracted child abuse and/or neglect can lead to biological changes, putting these individuals at greater risk for committing homicide and other forms of violence than those without child maltreatment histories. The implications of these biological findings for forensic evaluations are discussed. Based on new understanding of the effects of child maltreatment, the authors invite law and mental health professionals to rethink their notions of justice and offender accountability, and they challenge policymakers to allocate funds for research into effective treatment and for service delivery. [Author Abstract]

Keywords: Adolescents  Attachment  Brain Development  Child Abuse  Criminal Behavior  Child Neglect  Children  Criminal Responsibility  Forensic Evaluation  Homicide  Juvenile Offenders  Literature Review  Mitigating Factors  Murder  Neglect  Neuroendocrinology  Neurophysiology  Posttraumatic Stress Disorder  PTSD  Sociopathy  Survivors  Trauma  Violence  

Accuracy Verified: Yes

5. Grand, D., Hartung, J., & Bergmann, U. (2007, Novembro). Conversando com… David Grand, John Hartung, Uri Bergmann [Talking with ... David Grand, John Hartung, Uri Bergmann]. Apresentação no I Congresso Ibero-Americano de EMDR, Brasília, Brasil.

Language: Portuguese

Format: Conference

Abstract:
Nos últimos dez anos, as pesquisas sugerem que o Transtorno de Estresse Pós-Traumático (TEPT) seja uma desordem de humor, memória e integração somatossensorial. Estudos com neuroimagens e neuroendocrinologia têm demonstrado marcadores muito claros desse transtorno, destacando a compreensão de que o TEPT seja uma desordem neurobiológica e não o reflexo de fraqueza psicológica. Nesta apresentação examinaremos o processo de formação e consolidação da memória, a neurobiologia do TEPT e as neuroimagens correspondentes. A integração dessas três linhas ilustrarão os efeitos do TEPT sobre a regulação do humor, memória e integração somatossensorial.

Over the past decade, research suggests that Post-Traumatic Stressm Disorder (PTSD) is a disorder of mood, memory and somatosensory integration. Studies with neuroimaging and neuroendocrinology have markers demonstrated very clear that disorder, highlighting the understanding that PTSD is a neurobiological disorder and not the reflection of psychological weakness. In this presentation we will examine the process formation and consolidation of memory, neurobiology of PTSD and neuroimaging corresponding. The integration of these three lines illustrate the effects of PTSD on the regulation of mood, memory and somatosensory integration.

Keywords: Neurobiology  Posttraumatic Stress Disorder  PTSD  

Accuracy Verified: Yes

6. van der Kolk, B. A., Burbridge, J. A., & Suzuki, J. (1997, June). The psychobiology of traumatic memory:  Clinical implications of neuro imaging studies. Annals of the New York Academy of Sciences, 821, 99-113. doi:10.1111/j.1749-6632.1997.tb48272.x .

Language: English

Format: Journal

Abstract:
Whereas most patients with PTSD construct a narrative of their trauma over time, it is a characteristic of PTSD that sensory elements of the trauma itself continue to intrude as flashbacks and nightmares, altered states of consciousness in which the trauma is relived, unintegrated with an overall sense of self. Because traumatic memories are so fragmented, it seems reasonable to postulate that extreme emotional arousal leads to failure of the central nervous system (CNS) to synthesize the sensations related to the trauma into an integrated whole. Earlier models for a biological substrate of these phenomena have become rapidly outdated with the availability of new information derived from neuroimaging studies of patients with PTSD. The emerging body of knowledge from these studies has stimulated a gradual shift in emphasis away from the neurochemicals involved in the organisms' response to overwhelming threat to a focus on the neuronal filters concerned in the interpretation of sensory information: the interactions between the various parts of the CNS that process and interpret the meaning of incoming information, such as the amygdala, hippocampus, corpus callosum, anterior cingulte, and prefrontal cortex. [Text, p. 99]
Review Article: 58 references. This volume of the Annals was published as "Psychobiology of posttraumatic stress disorder," edited by Rachel Yehuda and Alexander C. McFarlane (New York: New York Academy of Sciences, 1997; ISBN 1-57331-078-6). These papers are the result of a conference entitled "Psychobiology of Posttraumatic Stress Disorder" sponsored by the New York Academy of Sciences, New York, 1996 September 7-10.

Keywords: Brain Imaging  Dissociative Amnesia  Etiology  Literature Review  Neuroanatomy  Neuroendocrinology  Psychobiology  PTSD  Stressors  Survivors  Treatment Effectiveness  

Accuracy Verified: Yes

7. Heber, R., Kellner, M., & Yehuda, R. (2002, December). Salivary cortisol levels and the cortisol response to dexamethasone before and after EMDR:  A case report. Journal of Clinical Psychology, 58(12), 1521-1530. doi:10.1002/jclp.10102.

Language: English

Format: Journal

Abstract:
Trauma survivors with PTSD have been shown to have lower basal cortisol levels in the urine, plasma, and saliva than in trauma survivors without PTSD, nontraumatized mentally ill, or healthy subjects. We report on a case study in which we measured pre- and post-Eye Movement Desensitization and Reprocessing (EMDR) treatment salivary cortisol levels and salivary cortisol response to 0.50 mg of dexamethasone in a 41-year-old female with chronic PTSD symptoms. Our goal was to determine whether symptom improvement following trauma-focused treatment (EMDR) is associated with changes in basal salivary cortisol or in the cortisol response to dexamethasone administration. Our findings show moderate symptom improvement, an increase in basal cortisol levels, and a more attenuated cortisol hypersuppression in response to the dexamethasone suppression test following EMDR treatment. These results suggest the potential utility of including neuroendocrine measures in the assessment of treatment outcome in PTSD. [Author Abstract]

Keywords: Battery  Biologic Markers  Brief Psychotherapy  Case Report  Clinical Case Study  Cortisol  Dexamethasone Suppression Test  Empirical Study  Females  Legal Procedures  Middle Aged  Multiple Traumatic Events  Neuroendocrine Neuroendocrine Testing  Neuroendocrinology  Posttraumatic Stress Disorder  PTSD  Rape  Survivors  Treatment Effectiveness  

Accuracy Verified: Yes