Francine Shapiro Library ~ EMDR Bibliography

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Your Results - you searched for the keyword Hypothalamic-Pituitary-Adrenal 11 Results

1. Gerardi, M., Rothbaum, B. O., Astin, M.C., & Kelley, M. (2010, June). Cortisol response following exposure treatment for PTSD in rape victims. Journal of Aggression, Maltreatment & Trauma, 19(4), 349-356. doi:10.1080/10926771003781297.

Language: English

Format: Journal

Abstract:
This study examined changes in salivary cortisol levels pre-to-post-treatment in adult female rape victims diagnosed with post traumatic stress disorder (PTSD) randomly assigned to be treated with either Prolonged Exposure Therapy or Eye Movement Desensitization and Reprocessing. Salivary cortisol was collected at baseline, session 3, and session 9. A significant decrease in salivary cortisol levels was observed in individuals classified as treatment responders in both treatment conditions. Findings suggest that successful exposure-based treatments for PTSD which result in trauma-related and depressive symptom reduction may impact the action of the hypothalamic-pituitary-adrenal axis as measured by changes in level of salivary cortisol from pre-to-post-treatment.

Keywords: Rape Posttraumatic Stress Disorder PTSD

Accuracy Verified: Yes

2. van der Kolk, B. A., & Stickgold, R. (1998, July). Current understanding of the psychobiology of trauma. Plenary presented at the annual meeting of the EMDR International Association, Baltimore, MD.

Language: English

Format: Conference

Abstract:
Participants will have basic knowledge about 1) the variety of psychophysiological responses to trauma; 2) current status of our knowledge about how the shutting off of the trauma response is mediated on hypothalamic-pituitary-adrenal axis, and the abnormalities of this biological system in PTSD; 3) basic understanding about the functions of different parts of the brain, in particular, the structure; and 4) explorations into possible modes of action of EMDR.

Keywords: Plenary Psychobiology

Accuracy Verified: Yes

3. Occhi, S., Albiol, L. M., & Cicognani, E. (2007). Il disturbo post-traumatico da stress: Una rassegna [Post-traumatic stress disorder: A review]. Psicoterapia Cognitiva e Comportamentale, 13(3), 323-344.

Language: Italian

Format: Journal

Abstract:
Questo articolo presenta una rassegna aggiornata teorico del Post-Traumatico da Stress Disorder (PTSD) concetto, un disturbo mentale che può comparire dopo essere stato esposto direttamente o indirettamente, ad un evento traumatico. L'articolo inizia con una descrizione della sintomatologia, suddivisi in: rivivere l'evento traumatico, evitamento, ottundimento reattività generale e ipervigilanza. Questo è seguito da una presentazione dei più recenti studi che indicano la presenza di disordine da stress post-traumatico nella popolazione, sia nella popolazione generale e nei soggetti direttamente coinvolti in un evento traumatico. Post-traumatico da stress sintomi del disturbo sono identificati mediante questionari specifici descritti nella sezione riferimento a strumenti di indagine. Lo studio delle basi biologiche per il PTSD è diventata particolarmente significativo negli ultimi anni, e abbiamo analizzato questo contesto in modo più approfondito per questo motivo, con particolare riferimento al ruolo dell'asse ipotalamo-ipofisi-surrene. Un impulso importante dello studio del PTSD è dovuto alla crescente importanza dato alla prevenzione, intesa come riduzione dell'impatto dei disturbi psichiatrici che concentrandosi su Eye Movement Desensibilizzazione e ritrattamento (EMDR), il trattamento psicologico, con il supporto di tecniche che riguarda la terapia cognitivo comportamentale e il trattamento farmacologico che prevede l'uso di inibitori della ricaptazione della serotonina selettiva, quali siano le forme più studiate. (PsycINFO record del database (c) 2008 APA, tutti i diritti riservati)

This article presents an updated theoretical review of the Post-Traumatic Stress Disorder (PTSD) concept, a mental disorder that can appear after being exposed directly or indirectly to a traumatic event. The article begins with a description of the symptomatology, divided in: re-living the traumatic event, avoidance, dulling general reactivity and hyperarousal. This is followed by a presentation of the most recent studies which indicate the presence of post-traumatic stress disorder in the population, both in the general population and in subjects directly involved in a traumatic event. Post-traumatic stress disorder symptoms are identified by using specific questionnaires described in the section referring to investigation instruments. The study of the biological bases for PTSD has become particularly significant in recent years, and we analysed this context in greater detail for this reason, with particular reference to the role of the hypothalamo-pituitary-adrenocortical axis. An important boost of the study of PTSD is due to the increasing importance placed on prevention, understood as a reduction of the impact of psychiatric disorders which focusing on Eye Movement Desensitisation and Reprocessing (EMDR), on psychological treatment, with the support of techniques that concerns cognitive behavioral therapy and pharmacological treatment that involves the use of serotonin selective reuptake inhibitors, which are the forms studied most. (PsycINFO Database Record (c) 2008 APA, all rights reserved)

Keywords: Posttraumatic Stress Disorder PTSD Review

Accuracy Verified: No

4. Bossini, L., Poliziotto, N., Tavanti, M., Calossi, S., Lombardelli, A., Vatti, G., & Castrogiovanni, P. (2006, Febbraio). Neuroimaging e PTSD: Dati morfovolumetrici e loro variazioni dopo trattamento [Neuroimaging and PTSD: Facts morfovolumetrici and their changes after treatment]. Presentazione al Congresso XI SOPSI (Società Italiana di Psicopatologia), Roma, Italia.

Language: Italian

Format: Conference

Abstract:
Introduzione: molti studi concordano sulla riduzione del volume ippocampale nei pazienti affetti da PTSD 1 e che tale alterazione anatomica è correlata con deficit cognitivi e con la gravità dei sintomi. Comunque ad oggi non è stato ancora chiarito se l’atrofia ippocampale rappresenta l’esito di un effetto neurotossico del trauma o, piuttosto, una condizione preesistente che predispone allo sviluppo di alcune patologie psichiatriche. Già da tempo studi su animali dimostrano come lo stress causi atrofia ippocampale e inibizione della neurogenesi, con meccanismi verosimilmente legati ai glucocorticoidi, all’increzione del fattore corticotropo, all’aumento degli aminoacidi eccitatori, all’inibizione fattore neurotrofico cerebrale con perdita della plasticità neuronale. Nell’uomo, tuttavia, i risultati non sono così lineari. Da un lato alcuni studi hanno individuato come fattore principale l’aumento dei glucocorticoidi 2, dall’altro tale teoria è stata fortemente criticata 3. Secondo Yehuda il meccanismo di atrofia ippocampale è dovuto ad un’alterazione dell’asse Ipotalamo-Ipofisi-Surrene (HPA), ma in termini di una bassa increzione di glucocorticoidi che determina un aumento del feedback negativo dell’asse stesso ed un’ipersensibilità recettoriale. Indipendentemente dal meccanismo d’azione, la perdita di neuroni a livello ippocampale nei soggetti che hanno subito eventi traumatici sembra sufficientemente dimostrata e, fino a poco tempo fa, era considerata irreversibile. In realtà l’ippocampo sembra presentare una inusuale e spontanea capacità rigenerativa. Questo dato è stato individuato in molte specie animali e, in un unico studio, anche nell’uomo 4. Inoltre recenti evidenze pre-cliniche e cliniche hanno indicato che gli SSRI (Selective Serotonin Reuptake Inhibitors) promuovono la neurogenesi e riducono l’atrofia ippocampale indotta dallo stress nell’animale 5 e nell’uomo sono in grado di ridurre i sintomi del PTSD, incrementare le dimensioni dell’ippocampo e ridurre i deficit mnesici tipici della patologia 6 7. Un altro fattore che sembra essere in grado di stimolare la neurogenesi negli animali sembra essere “l’ambiente arricchito” verosimilmente tramite i meccanismi molecolari dell’apprendimento che sembrano in grado di attivare la trascrizione dell’mRNA per il Brain Derived Neurotrophic Factor. Questo dato della letteratura supporta il razionale dell’efficacia della psicoterapia anche se l’unico studio che valuta le modificazioni morfostrutturali dopo psicoterapia non ha riportato risultati positivi 8. Gli scopi di questo studio sono: – valutare la presenza di atrofia ippocampale nei pazienti affetti da PTSD (T0-drug-free); – valutare l’effetto della terapia: farmacologica con SSRI e psicoterapica con EMDR (Eye Movement Desensitization and Reprocessing) sia sul piano clinico e neuropsicologico, che sul volume ippocampale, sia sulla memoria (T1). Metodologia: abbiamo analizzato un campione di 20 pazienti, di età compresa tra i 15 ed i 65 anni, reclutati nell’ambulatorio psichiatrico del Policlinico universitario di Siena affetti da PTSD e un gruppo di controllo di soggetti sani appaiati per sesso, età, peso e altezza. I soggetti di entrambi i gruppi sono stati sottoposti ad uno studio morfovolumetrico computerizzato dell’Ippocampo tramite RM (Risonanza Magnetica). Inoltre, i diciassette pazienti con PTSD sono stati valutati tramite la somministrazione di test neuropsicologici e scale psicometriche per approfondire il quadro psicopatologico e valutare l’eventuale presenza di deficit cognitivi. Nei soggetti affetti da PTSD dopo un periodo di sei mesi di terapia psicofarmacologica sono stati ripetuti i test neuropsicologici, le scale psicometriche e l’analisi morfovolumetrica dell’ippocampo tramite RM. Tre pazienti, dopo le valutazioni al T0, hanno effettuato un protocollo terapeutico con solo EMDR e sono stati rivalutati dopo 8 sedute (due mesi). Risultati: i risultati della prima parte sperimentale (T0-drug-free) evidenziano che le dimensioni dell’ippocampo di sinistra nei soggetti affetti da PTSD sono significativamente minori rispetto ai controlli sani. Dai risultati osservati al follow-up (T1-post-terapia) è possibile evincere che la terapia nei soggetti considerati è associata ad un miglioramento della sintomatologia e ad un aumento dei volumi ippocampali, pari al 9,87% per l’ippocampo di destra e dell’8,37% per l’ippocampo di sinistra. Questi dati sono concordi con i dati presenti in letteratura, anche se la percentuale di recupero su base neuroplastica nel nostro studio risulta sensibilmente superiore rispetto ai due studi presenti in letteratura incremento pari al 4,6% 6; pari al 5% 7. I tre pazienti che hanno effettuato terapia con EMDR hanno anch’essi mostrato al T1 un miglioramento sintomatologico (CAPS non più positiva per i criteri diagnostici) ed un aumento medio dei volumi ippocampali pari a 338,25 mm3 per l’ippocampo DX e 357,93 mm3 per l’ippocampo SN. Conclusioni: la terapia nei soggetti considerati si è associata ad un aumento dei volumi ippocampali (9,87%-8,37%). L’aumento dei volumi ippocampali appare rilevante, consistente con i dati in letteratura, sebbene quantitativamente superiore, sottolineando l’efficacia degli SSRI verosimilmente tramite il meccanismo di attivazione della neurogenesi; è ipotizzabile che l’aumento di volume non sia da imputare ad un aumento delle cellule gliali ma ad un aumento di neuroni ippocampali visto il contemporaneo miglioramento clinico. Particolarmente interessante ci sembra il dato relativo all’efficacia clinica e sulla plasticità neurale della EMDR. Questa osservazione su solo tre casi, necessita chiaramente di essere confermata su un campione più ampio ma rappresenta la prima evidenza in letteratura di un’azione della psicoterapia diretta alla struttura cerebrale.

Introduction: Many studies agree on the reduction of hippocampal volume in patients with PTSD and that an anatomical alteration is correlated with cognitive deficits and the severity of symptoms. However to date has not yet been clarified whether hippocampal atrophy is the result of a neurotoxic effect of trauma or, rather, an underlying condition that predisposes to the development of some psychiatric disorders. For some time animal studies show that stress causes hippocampal atrophy and inhibition of neurogenesis, by mechanisms probably related to glucocorticoids, all'increzione corticotropo factor, increased excitatory amino acid, inhibition of brain neurotrophic factor with loss of neuronal plasticity. In humans, however, the results are not so linear. On the one hand, some studies have identified as the main factor increasing glucocorticoid two other such theory was strongly criticized 3. According to Yehuda mechanism of hippocampal atrophy is due to an alteration of hypothalamic-pituitary-adrenal (HPA), but in terms of a low secretion of glucocorticoids leading to an increase of negative feedback axis and the same receptor hypersensitivity . Regardless of the mechanism of action, loss of neurons in hippocampus in people who have suffered traumatic events seems sufficiently established and, until recently, was considered irreversible. In fact, the hippocampus appears to be an unusual and spontaneous regenerative capacity. This figure has been identified in many animal species and in one study in humans 4. Moreover, recent evidence pre-clinical and clinical studies have shown that SSRIs (Selective Serotonin Reuptake Inhibitors) promote neurogenesis and reduce stress-induced hippocampal atrophy in animals 5 and humans are able to reduce symptoms of PTSD, increase the size of the hippocampus and reduce the deficit mnesic typical of the disease 6 7. Another factor that seems to be able to stimulate neurogenesis in animals seems to be "enriched environment" probably through molecular mechanisms of learning that seem able to activate the transcription of mRNA for Brain Derived Neurotrophic Factor. This finding supports the rationale of the literature of the effectiveness of psychotherapy, even if the only study that evaluates changes morphostructural after psychotherapy has shown positive results 8. The aims of this study are: - To evaluate the presence of hippocampal atrophy in patients with PTSD (T0-drug-free) - to assess the effect of therapy: pharmacological SSRI and psychotherapy with EMDR (eye movement desensitization and reprocessing) is a clinical and neuropsychological, and on hippocampal volume, and memory (T1). Methods: We analyzed a sample of 20 patients, aged between 15 and 65, recruited nell'ambulatorio Psychiatric University Hospital of Siena with PTSD and a control group of healthy subjects matched by sex, age, weight and height. Subjects in both groups were subjected to a computerized study morfovolumetrico dell'Ippocampo using MRI (Magnetic Resonance). In addition, seventeen patients with PTSD were assessed through administration of psychometric scales and neuropsychological tests to study the psychopathological picture and evaluate the possible presence of cognitive deficits. In subjects with PTSD after a period of six months of pharmacological therapy were repeated neuropsychological tests, scales psychometric analysis morfovolumetrica hippocampus by MRI. Three patients at T0 after assessments, carried out a treatment protocol with only EMDR and were reassessed after eight sessions (two months). Results: The results of the first experiment (T0-drug-free) show that the size of the left hippocampus in patients with PTSD are significantly lower compared to healthy controls. The results observed during the follow-up (T1-post-therapy) can be inferred that therapy in patients considered to be associated with improvement in symptoms and an increase in hippocampal volume, equal to 9.87% for the right hippocampus and 8, 37% for the left hippocampus. These data are consistent with the data in the literature, although the recovery rate based on neuroplastic in our study is significantly higher than in the two studies in the literature increase of 4.6% 6; 5% 7. The three patients who have treatment with EMDR have also shown an improvement in symptoms at T1 (CAPS no longer positive for the diagnostic criteria) and an average increase in hippocampal volume amounted to 338.25 mm3 for the hippocampus and DX 357, 93 mm3 for the hippocampus SN. Conclusions: Therapy in patients considered was associated with an increase in hippocampal volume (9.87% -8.37%). The increase in hippocampal volume appear to be relevant, consistent with the literature data, although quantitatively greater, stressing the effectiveness of SSRIs probably through the mechanism of activation of neurogenesis, it is conceivable that the increase in volume is attributable to an increase glial cells but an increase of hippocampal neurons seen the simultaneous clinical improvement. Seems particularly interesting given the relative clinical effectiveness of EMDR and neural plasticity. This observation on only three cases, clearly needs to be confirmed on a larger sample but represents the first evidence in the literature of action of psychotherapy directed at brain structure.

Keywords: Posttraumatic Stress Disorder PTSD

Accuracy Verified: Yes

5. Davidson, J. R. T., Stein, D. J., Shalev, A.Y., & Yehuda, R. (2004, Spring). Posttraumatic stress disorder: Acquisition, recognition, course, and treatment. Journal of Neuropsychiatry and Clinical Neuroscience, 16(2), 135-147. doi:10.1176/appi.neuropsych.16.2.135.

Language: English

Format: Journal

Abstract:
Following exposure to trauma, a large number of survivors will develop acute symptoms of posttraumatic stress disorder (PTSD), which mostly dissipate within a short time. In a minority, however, these symptoms will evolve into chronic and persistent PTSD. A number of factors increase the likelihood of this occurring, including characteristic autonomic and hypothalamic-pituitary-adrenal axis responses. PTSD often presents with comorbid depression, or in the form of somatization, both of which significantly reduce the possibilities of a correct diagnosis and appropriate treatment. Mainstay treatments include exposure-based psychosocial therapy and selective serotonin reuptake inhibitors, such as paroxetine and sertraline, both of which have been found to be effective in PTSD. This paper looks at the course of PTSD, its disabling effect, its recognition and treatment, and considers possible new research directions.

Keywords: Comorbidity Posttraumatic Stress Disorder PTSD Recognition and Treatment SSRIs

Accuracy Verified: Yes

6. Ross, C. (2010, Septembrer/October). Psychobiology of dissociation. Plenary presented at the annual meeting of EMDR International Association, Minneapolis, MN.

Language: English

Format: Conference

Abstract:
The session will: explain four different meanings of the word “dissociation" and clarify which ones are relevant to the psychobiology of dissociation; review the structural model of dissociation and how it unifies the dissociative disorders and PTSD; and describe examples of dissociations between declarative and procedural memory from neurology and experimental cognitive psychology. Most of the session will focus on existing and future research on the biology of dissociation from a trauma perspective. Data on hippocampal volume reduction, the hypothalamic-pituitary-adrenal axis and other biological systems will be reviewed.

Keywords: Dissociation Plenary Psychobiology

Accuracy Verified: Yes

7. Leserman, J. (2005). Sexual abuse history: Prevalence, health effects, mediators, and psychological treatment. Psychosomatic Medicine, 67(6), 906-915. doi:10.1097/01.psy.0000188405.54425.20.

Language: English

Format: Journal

Abstract:
Objective: Lifetime history of sexual abuse is estimated to range between 15% and 25% in the general female population. People who are sexually abused are at greater risk for a whole host of physical health disorders that may occur many years after the abusive incident(s). Despite the high prevalence of this trauma and its association with poor health status, abuse history often remains hidden within the context of medical care. The aims of this review are to determine which specific health disorders have been associated with sexual abuse in both women and men, to outline the types of sexual abuse associated with the worst health outcome, to discuss some possible explanations and mediators of the abuse/health relationship, to discuss when and how to talk about abuse within a clinical setting, and to present evidence for which psychological treatments have been shown to improve the mental health of patients with past sexual abuse. Method: To meet these objectives, we have reviewed a wide literature on the topic of sexual abuse. Results: We demonstrate that abuse appears to be related to greater likelihood of headache and gastrointestinal, gynecologic, and panic-related symptoms; that the poor health effects associated with abuse are also seen in men; that abuse involving penetration and multiple incidents appears to be the most harmful, and that exposure-type therapies with and without cognitive behavioral therapy hold promise for those with abuse history. Conclusion: We need more research examining psychological treatments that might be efficacious in treating the physical health problems associated with sexual abuse history.

Keywords: HMO Health Maintenance Organization HPA Hypothalamic-Pituitaryadrenocortical Review Posttraumatic Stress DIsorder PTSD Sexual Abuse Trauma

Accuracy Verified: Yes

8. Chivers-Wilson, K. A. (2006). Sexual assault and posttraumatic stress disorder: A review of the biological, psychological and sociological factors and treatments. McGill Journal of Medicine, 9(2), 111-118.

Language: English

Format: Journal

Abstract:
Sexual assault occurs with alarming frequency in Canada. The prevalence of Posttraumatic Stress Disorder (PTSD) in assault survivors is drastically higher than the national prevalence of the disorder, which is a strong indication that the current therapies for sexualassault- related PTSD are in need of improvement. Increasing knowledge and understanding of the pathologies associated with rape trauma in biological, psychological and sociological domains will help to develop more effective treatments for survivors. A dysregulation of the Hypothalamic- Pituitary-Adrenal (HPA) axis is observed in survivors of sexual assault and this may be a fundamental cause of the structural and functional abnormalities contributing to PTSD symptoms. Pharmacotherapies are available to treat PTSD; however, they are often inadequate or unwanted by the survivor. Psychological health is compromised following interpersonal trauma and many psychological therapies are available, but with varying efficacy. A person's cognitions have a dramatic effect on the onset, severity, and progress of PTSD following sexual assault. Sociological impacts of assault influence the development of PTSD through victim-blaming attitudes and the perpetuation of rape myths. Perceived positive regard and early social support is shown to be important to successful recovery. Education is vital in rape prevention and to foster a supportive environment for survivors. The biological, psychological and sociological impacts and treatments should not remain mutually exclusive. A better appreciation of the biopsychosocial repercussions of sexual assault will aid in developing a more holistic and individualized therapy to help alleviate the physical and emotional pain following the trauma of rape.

Keywords: Pharmacotherapy Posttraumatic Stress Disorder PTSD Rape Sexual Assault Trauma

Accuracy Verified: Yes

9. Morton, B. E., & Paulsen, S. L. (1993). Stress disorders: Cortical plasticity, eye saccades, and multiple personalities. Paper presented at the University of Hawaii School of Medicine and Pacific Institute for Behavioral Medicine. Honolulu, HI.

Language: English

Format: Other

Abstract:
Origin and Nature of Stress Disorders: The Defense Response occurs as part of the switch from fearful flight (Ego) to cornered, fearless fight (Id) whereby the individual attempts to obtain freedom from restraint through the violence of defensive attack. The activation of the hypothalamic-pituitary-adrenal (HPA) axis is part of this response. Stress Disorders are pathological states resulting in inappropriate, repeated activation of the Defense Response. They appear to be produced from a maladaptive cortical plasticity that occurred whenever the Defense Response failed to protect the organism from trauma.

Keywords: Cortical Plasticity Defense Response Eye Saccades HPA Hypothalamic-Pituitary-Adrenal Multiple Personalities Stress Disorders

Accuracy Verified: Yes

10. Unfried, N. (2003). Trauma und entwicklung: Physiologische und biologische veränderungen nach frühen kindlichen traumata und deren behandlungsmöglichkeit [Trauma and development: Physiologic and biologic variations after early infant traumatisations and attendance of them]. Zeitschrift für Psychotraumatologie und Psychologische Medizin (ZPPM), 1(3), 59-71.

Language: German

Format: Journal

Abstract:
Behandlung psychotraumatischer belastungsstörungen mit EMDR
Die Entwicklung des Kindes ist heutzutage als Prozess zu verstehen. Ein Kind ist dementsprechend zu jedem Zeitpunkt seiner Entwicklung "reif", einschließlich seines intrauterinen Lebens, d.h. es verfügt über die für die jeweilige Zeit notwenige Ausstattung. Von Beginn an erfolgt dieses mehr oder weniger störanfällige Geschehen mit anderen im aktiven intra- und interagierenden informativen, energetischen und stofflichen Austausch. An diesem Entwicklungsprozess nimmt der gesamte Körper, jede Zelle, einschließlich des Gehirns als Organ der sensomotorischen und psychophysischen Verarbeitung teil. Die Stressreaktion und Stress auf bewältigbarem Niveau hilft dem Kind kritische Phasen zu überstehen (Hüther, 1999). Jedoch führt nicht bewältigbarer Stress zu tiefgreifenden Veränderungen funktionell bis strukturell, wenn der Organismus keine neue Lösungsmöglichkeit findet. Mehrere Autoren belegen, dass traumatische Erlebnisse Veränderungen im limbischen System und Cortex zeigen können (Hüther, 1999; van den Kolk, 1998; Roth, 1998). Bei unkontrolliertem Stress (frühzeitig) kommt es zur Daueraktivierung der Amygdala und über die Amygdala zur Aktivierung mehrerer Systeme, unter anderem auch der Hypothalamus-Hypophysen-Nebennieren- Achse mit einem Ausschütten von Stresshormonen. Diese Daueraktivierung löst körperlich die Notfallreaktion im Sinne einer Schockreaktion aus, gleichzeitig führt sie zur Störung der Einspeicherung von Informationen in den Hippocampus. Die imaginativ-methodische Herangehensweise scheint für frühtraumatisierte Kinder und Jugendliche eine Möglichkeit zu sein, die dissoziierten Anteile der traumatischen Szene abzurufen und somit einen Weg zur Integration zu finden. Die therapeutische Beziehung ermöglicht das Wiedererleben der Schmerzen, der Angst, aber auch die Beendigung der traumatischen Situation. Es konnte gezeigt werden, wie die triggerabhängigen Projektionen bei den Kindern endeten und Veränderungen der Persönlichkeitsentwicklung nachweisbar waren. Der Erfahrungsbericht stellt ein vorläufiges Ergebnis dar.

Psychosocial treatment of traumatic stress disorders with EMDR
Children’s development is now understood as a process. Balance and imbalance are said to alternate with one another, and impaired functioning is to be seen as an inducement for further development. Even the early organism has the opportunity of finding a new level of organisation. Right from the beginning, this process, which is susceptible to disruption to a greater or lesser extent, takes place with others in an intra- and interactive exchange of energy and material. This developmental process involves the entire body, every cell, including the brain as the organ of sensomotoric and psychophysical processing. The stress reaction and stress at a manageable level help the child to survive critical periods (Hüther, 1999). However, stress that is not manageable leads to far-reaching changes, in both functional and structural terms, unless the organism finds new solutions. There are sensitive stages during prenatal development that give the brain a high degree of adaptability; however, they also make the embryo, foetus and young infant receptive for disruptive or even hostile influences can lead to changes in the limbic system and the cortex (Hüther 1999; van den Kolk 1998; Roth, 1998). In the event of (early) uncontrolled stress, the amygdala becomes permanently activated, and via the amygdala, several systems are also activated, including the hypothalamic-pituitary-adrenal axis, by the secretion of stress hormones. This permanent activation triggers a physical emergency reaction in the sense of a shock reaction and at the same time leads to a disruption of the storage of information in the hippocampus. The imaginative approach to be a way for children and adolescents with early traumas to recall the dissociated parts of the traumatic scene and hence to find a way of integrating them. The therapeutic relationship allows the pain and fear to be reenacted, but also enables the traumatic situation to be brought to a close. It was able to be shown how the trigger-dependent projections stopped in the children, and changes in personality development were able to be observed. The report presents preliminary results.

Keywords: Attachment Chidlren Biologic Variations Psysiologic Variations Trauma

Accuracy Verified: Yes

11. Schore, A. (2000, September). Traumatic attachment and the development of the right brain. Presentation at the annual meeting of the EMDR International Association, Toronto, Ontario Canada.

Language: English

Format: Conference

Abstract:
Participants will learn: 1) how a history of early abuse and/or neglect induces severe attachment pathology in infants and toddlers; 2) the deleterious effects of early abuse and neglect on the development of right brain coping systems, specifically focusing on the impairment of the maturation of higher right coritcal interconnections into the hypothalamo-pituitary-adrenal axis and the sympthomedullary axis; and 3) a developmental psychoneurobiological model of dissociation.

Keywords: Attachment Dissociation Right Brain Trauma

Accuracy Verified: Yes