Francine Shapiro Library ~ EMDR Bibliography

Francine Shapiro Library - EMDR Bibliography Links

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Your Results - you searched for the keyword Cortisol 13 Results

1. Aurora, R. N., Zak, R. S., Auerbach, S. H., Casey, K. R., Chowdhuri, S., Karippot, A., Maganti, R. K., Ramar, K., Kristo, D. A., Bista, S. R., Lamm, C. I., & Morgenthaler, T. I. (2010, August). Best practice guide for the treatment of nightmare disorder in adults. Journal of Clinical Sleep Medicine, 6(4), 389-401.

Language: English

Format: Journal

Abstract:
Prazosin is recommended for treatment of Posttraumatic Stress Disorder (PTSD)-associated nightmares. Level A. Image Rehearsal Therapy (IRT) is recommended for treatment of nightmare disorder. Level A. Systematic Desensitization and Progressive Deep Muscle Relaxation training are suggested for treatment of idiopathic nightmares. Level B. Venlafaxine is not suggested for treatment of PTSD-associated nightmares. Level B. Clonidine may be considered for treatment of PTSD-associated nightmares. Level C. The following medications may be considered for treatment of PTSD-associated nightmares, but the data are low grade and sparse: trazodone, atypical antipsychotic medications, topiramate, low dose cortisol, fluvoxamine, triazolam and nitrazepam, phenelzine, gabapentin, cyproheptadine, and tricyclic antidepressants. Nefazodone is not recommended as first line therapy for nightmare disorder because of the increased risk of hepatotoxicity. Level C. The following behavioral therapies may be considered for treatment of PTSD-associated nightmares based on low-grade evidence: Exposure, Relaxation, and Rescripting Therapy (ERRT); Sleep Dynamic Therapy; Hypnosis; Eye-Movement Desensitization and Reprocessing (EMDR); and the Testimony Method. Level C. The following behavioral therapies may be considered for treatment of nightmare disorder based on low-grade evidence: Lucid Dreaming Therapy and Self-Exposure Therapy. Level C No recommendation is made regarding clonazepam and individual psychotherapy because of sparse data.

Keywords: Nightmares Posttruamatic Stress Disorder PSTD

Accuracy Verified: Yes

2. van der Kolk, B. A., Hopper, J., & Spinazzola, J. (2004, November). Biological changes in arousal and cortisol following PTSD treatment. Symposium conducted (M. Olff, Chair) at the 20th annual meeting of the International Society of Traumatic Stress Studies, New Orleans, LA.

Language: English

Format: Conference

Abstract:
Effects of treatment of PTSD on psychobiological measures: It is well known that PTSD is associated with changes in several biological systems. However little research has been done on whether it is possible to “reset” these biological systems with effective psychotherapy of pharmacological therapy. This symposium will present data on neuroendocrine and neuroimaging outcome measures.
Biological changes in arousal and cortisol following PTSD treatment: This presentation will discuss the results of a controlled treatment outcome study comparing EMDR, fluoxetine and pill placebo and demonstrate how effective treatment resulted in changes in memory processes, utilizing the Traumatic Memory Inventrory. In the EMDR condition, but not fluoxetine, the change in traumatic memory towards an integrated narrative was linearly correlated with physiological arousal in response to script driven imagery. This presentation will also present the relationship between clinical improvement in the three conditions and change in the cortisol response to a dexamethasone challenge.

Keywords: Fluoxetine Pill Placebo Posttraumatic Stress Disorder PSTD Symposium

Accuracy Verified: Yes

3. Resick, P., Monson, C., Griffin, M., Rothbaum, B., Rasmusson, A., & Shalev, A. (2006, November). Cortisol pre and posttreatment with EMDR or prolonged imaginal exposure in PTSD assault survivors. In Psychobiology and Treatment of PTSD. Symposium conducted at the 22nd annual meeting of the International Society for Traumatic Stress Studies Fall Conference, Hollywood, CA.

Language: English

Format: Conference

Abstract:
Psychobiological treatment of PTSD: This symposium will examine four CBT treatment studies with regard to biological markers. The questions here are whether pretreatment psychobiology or physiological responding can be used to predict treatment outcome, or whether they themselves change as a result of effective treatment.

Cortisol pre and posttreatment with EMDR or prolonged imaginal exposure in PTSD assault survivors: Many studies have noted increased cortisol production in trauma survivors with PTSD, but it is not clear whether effective treatment alters these responses. As part of a larger study, 60 female sexual assault survivors with PTSD began one of two types of cognitivebehavioral treatment (Prolonged Exposure (PE) or EMDR). Each treatment consisted of nine sessions. Sessions 1 and 2 included information gathering, trauma education, and therapy preparation. Sessions 3 through 9 consisted of processing traumatic memories and emotions via either imaginal exposure or EMDR.To examine potential cortisol changes over the course of treatment, salivary cortisol samples were collected at three time points during treatment. A baseline sample was taken at session 1, a second sample was taken at the start of the treatment portion of therapy (session 3), and a third sample was taken at the end of treatment (session 9). Of the original sample of 60 participants, 50 women completed treatment, and ten dropped out. Cortisol responses will be examined in treatment responders and non-responders as well as in treatment completers vs. treatment dropouts.

Keywords: Cortisol Posttraumatic Stress Disorder Prolonged Imaginal Exposure Assault PSTD Survivors Symposium

Accuracy Verified: Yes

4. Rothbaum, B. O., Astin, M., Gerardi, M., & Kelley, M. (2006, November). Cortisol pre-and post- treatment with EMDR or prolonged imaginal exposure in PTSD assault survivors. Presentation at the Annual Meeting of the Internatinal Society for Traumtic Stress Studies, Hollywood, CA.

Language: English

Format: Conference

Keywords: Assault Posttraumatic Stress Disorder Prolonged Imaginal Exposure PTSD Survivors

Accuracy Verified: Yes

5. Gerardi, M., Rothbaum, B. O., Astin, M.C., & Kelley, M. (2010, June). Cortisol response following exposure treatment for PTSD in rape victims. Journal of Aggression, Maltreatment & Trauma, 19(4), 349-356. doi:10.1080/10926771003781297.

Language: English

Format: Journal

Abstract:
This study examined changes in salivary cortisol levels pre-to-post-treatment in adult female rape victims diagnosed with post traumatic stress disorder (PTSD) randomly assigned to be treated with either Prolonged Exposure Therapy or Eye Movement Desensitization and Reprocessing. Salivary cortisol was collected at baseline, session 3, and session 9. A significant decrease in salivary cortisol levels was observed in individuals classified as treatment responders in both treatment conditions. Findings suggest that successful exposure-based treatments for PTSD which result in trauma-related and depressive symptom reduction may impact the action of the hypothalamic-pituitary-adrenal axis as measured by changes in level of salivary cortisol from pre-to-post-treatment.

Keywords: Rape Posttraumatic Stress Disorder PTSD

Accuracy Verified: Yes

6. Capezzani, L. (2010, Novembre). Dati preliminari del progetto: Valutazione degli esiti medico- clinici e psicologici in seguito all’applicazione dell’EMDR in pazienti oncologici con disturbi dello spettro post traumatico da stress [Preliminary data of the project: Evaluation of medical-clinical and psychological outcomes following the application of EMDR in cancer patients with autism spectrum post-traumatic stress disorder]. Presentazione al "Convegno La psicotraumatologia Oncologica, Roma, Italia.

Language: Italian

Format: Conference

Abstract:
La ricerca ha lo scopo di indicare se l’intervento con EMDR produce significativi cambiamenti dei parametri medico-clinico, oltre che psicologici, in pazienti oncologici che abbiamo ricevuto una diagnosi di PTSD o di un disturbo dello spettro post-traumatico da stress durante ciascuna delle fasi dell’evoluzione della malattia. In particolare si vuole verificare se dopo trattamento con EMDR: - i valori baseline di cortisolo nel sangue cambiano e riproducono anche in pazienti oncologici l’andamento dimostrato in letteratura: in presenza di un PTSD acuto generalmente i livelli sono alti, quando invece il PTSD è cronico i livelli di cortisolo sono bassi ma in ambedue i casi un trattamento con EMDR produce una normalizzazione di suddetti valori. - si osserva una riduzione della quantità di citochine che rappresentano gli indicatori immunologici correlati a stati di depressione ed ansia sia sottosoglia che non, quasi sempre presenti tra i disturbi dello spettro PTSD e quindi anche nella malattia oncologica (Cantelmi, 2008 in preparazione). Lo studio consente inoltre di osservare - per quali delle fasi della malattia il trattamento con EMDR produce la migliore estinzione dei disturbi post-traumatici da stress, - se le modalità di coping, cioè le strategie di adattamento alla malattia e sua gestione migliorano dopo il trattamento con EMDR

The research is intended to indicate whether the intervention with EMDR produces significant changes in clinical parameters and medical as well as psychological, in cancer patients who have received a diagnosis of PTSD or a spectrum disorder post-traumatic stress disorder during each of the stages of the disease. In particular, we want to check if after treatment with EMDR: - The values of baseline cortisol in the blood change and reproduce even in cancer patients the trend shown in the literature: in the presence of an acute PTSD generally the levels are high, when instead the PTSD is chronic cortisol levels are low but in both cases treated with EMDR produces a normalization of these values. - There is a reduction in the amount of cytokines representing the immunological indicators related to states of depression and subthreshold anxiety is that, almost always present between the spectrum disorders PTSD and therefore also in the oncological disease (Cantelmi, 2008 in preparation). The study also allows you to observe - For which of the stages of the disease treatment with EMDR produces the best extinction of the symptoms of post-traumatic stress, - Whether the method of coping, ie the strategies of adaptation to the disease and its management to improve after treatment with EMDR.

Keywords: Autism Cancer Posttraumatic Stress Disorder PTSD

Accuracy Verified: Yes

7. Softic, R, & Becirovic, E, (2009, Septembra). Desenzitizacija pokretima ociju i reprocesiranje (EMDR): Kada su rijeci nemocne [Desensitization of eye movements and reprocessing (EMDR): When words are disabled]. Prvi Psihoterapijski Simpozijum Bosne I Hercegovine Sarajevo, Bosnia and Herzegovina.

Language: Serbian

Format: Conference

Abstract:
Neki ljudi doţive traumatska stanja koja ne mogu u potpunosti opisati rijeĉima. Mnogi od njih nikada ni ne progovore o tome što su doţivjeli. Ali patnja se u njima nastavlja i organizam, a ĉesto i okolina plaćaju ogromnu cijenu. U takvim sluĉajevima psihoterapija koja koristi rijeĉi je nemoćna. Istraţivanja upućuju da jedna, relativno nova psihoterapijska metoda desenzitizacija pokretima oĉiju i reprocesiranje (EMDR), moţe pomoći i takvim osobama. Kada neko iskusi ozbiljnu psihološku traumu, ĉini se da dolazi do narušavanja ravnoteţe u nervnom sistemu. Ta ravnoteţa je, moguće, narušena i posredstvom razliĉitih medijatora poput adrenalina, serotonina, dopamina, kortizola itd. Zbog takve neravnoteţe onemogućeno je optimalno funkcioniranje sistema za procesiranje informacija, a informacije vezane uz traumatiĉan dogaĊaj poput slika, zvukova, afekata i fiziĉkih senzacija se zadrţavaju u disfunkcionalnom, uznemirujućem obliku. Danas izranja shvatanje da postoji неуролошки balans u razliĉitim fiziološkim sistemima što omogućuje da informacije budu procesirane na adaptivan naĉin. EMDR kod nekih traumatiziranih djeluje ĉak i kada osoba ne moţe govoriti o tome što je preţivjela. Ono što je bitno jeste da se fokusira na traumatski dogaĊaj i da suraĊuje sa terapeutom u procesu stimulisanja dualne paţnje što omogućava adaptivno procesiranje informacija. Adaptivno procesiranje znaĉi uspostavljanje adekvatnih asocijacija i pojavu da iskustvo biva konstruktivno ugraĊeno u pozitivne kognitivne i emocionalne sheme pojedinca. Odnosno, oslobaĊa od simptoma i omogućava da se nova iskustva doţive bez blokirajućeg uticaja traume.

Some people doţive traumatic conditions that can not fully describe in words. Many of them never to speak of it as doţivjeli. But the suffering in them continues and the body, and environment, and often pay a huge price. In such cases psychotherapy that uses words of the powerless. Research suggests that a relatively new psychotherapeutic methods desensitization and reprocessing eye movement (EMDR), and can help such people. When someone has experienced serious psychological trauma, it seems that there is a violation of equilibrium in the nervous system. This equilibrium is possible, and disrupted through a variety of mediators such as adrenaline, serotonin, dopamine, cortisol, etc.. Because of such imbalance prevented the optimal functioning of the system for information processing and information related to traumatic an event such as images, sounds, physical sensations and affects it retains in the dysfunctional, disturbing form. It emerges that there is understanding of neurological balance in different physiological systems, which allows information to be processed on adaptive manner. EMDR works by some traumatized even when the person can not talk about what is preţivjela. What is important is to focus on the traumatic events and to co-operate with the therapist in the process of stimulating the attention of the dual provides adaptive information processing. Adaptive processing means getting the right associations and the emergence of the experience of being a built-in positive constructive cognitive and emotional schemas of the individual. That is, oslobaĊa of symptoms and allows for new experiences doţive without blocking effects of trauma.

Keywords: Doţ ivjeli Preţ ivjela

Accuracy Verified: Yes

8. Hopper, J., Spinazzola, J., Blaustein, M., Yehuda, R., van der Kolk, B. A., & Simpson, W. (2003, October-November). Differential biological outcomes of EMDR and fluoxetine for PTSD. In B. A. van der Kolk (Chair), Treatment outcome studies of PTSD. Symposium conducted at the 19th annual meeting of the International Society for Traumatic Stress Studies, Chicago, IL.

Language: English

Format: Conference

Abstract:
Treatment Outcome Studies of PTSD: This symposium presents three large carefully controlled treatment outcome studies using four different treatment modalities (CBT, EMDR, psychopharmacology and Cognitive Processing) and presents data on comparative efficacy, treatment responsiveness and resistance, effects on comorbidity, quality of life, and biological changes that accompany symptom improvement.

Differential Biological Outcomes of EMDR and Fluoxetine for PTSD: Two major developments in PTSD research have been the development of effective treatments and the delineation of pathophysiology. While major gains have been made in both of these areas, they have occurred in parallel. There is a need for investigations of possible differential effects of different treatment modalities on biological aspects of PTSD. In this randomized controlled trial (N = 105), we compared the exposure treatment Eye Movement Desensitization and Reprocessing, the serotonergic reuptake inhibitor fluoxetine, and pill placebo for their effects on both PTSD symptoms and biological parameters. The outcome measures were severity of reexperiencing, avoidant/numbing, and hyperarousal symptoms; psychophysiological reactivity to script-driven imagery; basal salivary cortisol and dexamethasone suppression test (DST). At pre-treatment, post-treatment and 3-month follow-up, saliva samples were acquired at 8 a.m., 11 p.m. (immediately followed by dexamethasone) and 8 a.m.. Participants also underwent a script-driven imagery protocol utilizing four 30s scripts, each followed by a 60s script imaging period and 2 minute recovery periods (fixed order: neutral, trauma, neutral, trauma). Preliminary analyses partially support hypothesizes concerning differential efficacies of pharmacological and psychological treatments on different symptom clusters and biological markers of the disorder, at post-treatment and 3- month follow-up. Potential implications for treatment and future research will be discussed.

Keywords: Fluoxetine Posttraumatic Stress Disorder PTSD Symposium

Accuracy Verified: Yes

9. Welch, K. L. (2007, August). EMDR and neuroscience research: Some questions and implications for psychotherapy integration. EMDR Practitioner. Retrieved from http://www.emdr-practitioner.net on 12/27/2008.

Language: English

Format: Other

Abstract:
Since its introduction, Eye Movement Desensitization and Reprocessing (EMDR) (Shapiro, 1989) has received the attention of many mental health professionals. There has been much critical debate on the subject of EMDR. Most of the clinical discussion has centered on the role of EMDR in the treatment of Posttraumatic Stress Disorder (PTSD). While the EMDR procedure has been compared to Mesmerism (McNally, 1999), declared as pseudoscience (Herbert, Lilienfeld, Lohr, Montgomery, O’Donohue, Rosen, and Tolin, 2000), or regarded as a highly marketed placebo (Lilienfield, 1996), most studies support the efficacy of EMDR in treating PTSD (Ironson, Freund, Strauss, and Williams, 2002; Lee, Gavriel, Drummond, Richards, and Greenwald, 2002; Marcus, Marquis, and Sakai, 1997; Rothbaum, 1997; Van Etten and Taylor, 1998; Wilson, Becker, and Tinker, 1997). There has been some evidence for accompanying physiological changes in PTSD subjects treated with EMDR with patterns of cortex functioning, (Levin, Lazrove, and van der Kolk, 1999; Nicosia, 1994) event-related potential changes (Lamprecht, Kohnke, Sack, Matzke and Munte, 2004), as well as positive effects on the level of the stress hormone cortisol (Haber, Kellner and Yehuda, 2002).

Keywords: Neuroscience

Accuracy Verified: Yes

10. Heber, R., Kellner, M., & Yehuda, R. (2002, December). Salivary cortisol levels and the cortisol response to dexamethasone before and after EMDR: A case report. Journal of Clinical Psychology, 58(12), 1521-1530. doi:10.1002/jclp.10102.

Language: English

Format: Journal

Abstract:
Trauma survivors with PTSD have been shown to have lower basal cortisol levels in the urine, plasma, and saliva than in trauma survivors without PTSD, nontraumatized mentally ill, or healthy subjects. We report on a case study in which we measured pre- and post-Eye Movement Desensitization and Reprocessing (EMDR) treatment salivary cortisol levels and salivary cortisol response to 0.50 mg of dexamethasone in a 41-year-old female with chronic PTSD symptoms. Our goal was to determine whether symptom improvement following trauma-focused treatment (EMDR) is associated with changes in basal salivary cortisol or in the cortisol response to dexamethasone administration. Our findings show moderate symptom improvement, an increase in basal cortisol levels, and a more attenuated cortisol hypersuppression in response to the dexamethasone suppression test following EMDR treatment. These results suggest the potential utility of including neuroendocrine measures in the assessment of treatment outcome in PTSD. [Author Abstract]

Keywords: Battery Biologic Markers Brief Psychotherapy Case Report Clinical Case Study Cortisol Dexamethasone Suppression Test Empirical Study Females Legal Procedures Middle Aged Multiple Traumatic Events Neuroendocrine Neuroendocrine Testing Neuroendocrinology Posttraumatic Stress Disorder PTSD Rape Survivors Treatment Effectiveness

Accuracy Verified: Yes

11. Mastronardi, C. (2007). Trauma da abuso e EMDR: Presentazione di una ricerca in corso [Abuse trauma and EMDR: A presentation of current research]. Universita Degli Studi di Milano, Italia.

Language: Italian

Format: Dissertation/Thesis

Abstract:
La tesi “Trauma da abuso e EMDR: presentazione di una ricerca in corso” mira ad esplorare il trauma in un’ottica sia psicologica che neurobiologica. Recenti ricerche dimostrano che i sopravvissuti affetti da Disturbo Post-traumatico da Stress (PTSD) non solo manifestano dei sintomi post traumatici – come intrusività, evitamento ed arousal – ma hanno anche livelli più bassi di cortisolo rispetto ai sopravvissuti senza Disturbo Posttraumatico da Stress. In questa tesi viene riportato uno studio su un caso singolo nel quale sono stati misurati i livelli di cortisolo urinario pre e post-trattamento EMDR (Eye Movement Desensitization and Reprocessing) in una ragazzina di quindici anni affetta da Disturbo Post-traumatico da Stress. L’obiettivo è determinare se il trattamento produce un decremento dei sintomi, misurati attraverso una valutazione testistica, e se a questo decremento è associato un aumento dei livelli di cortisolo urinario. I risultati indicano una diminuzione dei sintomi che ha comportato una remissione del PTSD. Questo miglioramento è confermato anche da un aumento dei livelli di cortisolo urinario. Questi risultati confermano l’efficacia dell’EMDR (trattamento evidence-based) per la cura del Disturbo Post-traumatico da Stress e suggeriscono l’utilità di includere misurazioni neuroendocrine nella valutazione dei risultati.

The thesis “Trauma da abuso e EMDR: presentazione di una ricerca in corso” aims at exploring the trauma from a psychological and neurobiological point of view. According to the latest studies, trauma survivors with posttraumatic stress disorder (PTSD) have been shown to have not only posttraumatic symptoms - as Avoidance, Intrusion and Arousal - but also lower basal cortisol levels than in trauma survivors without PTSD. In this thesis a single case study, in which pre and post–Eye Movement Desensitization and Reprocessing (EMDR) treatment urinary cortisol levels have been measured in a 15-year old female with PTSD, has been reported. The aim of this study is the assessment of the EMDR treatment effectiveness in symptom reduction measured with psychological instruments; besides, the assessment of the changes in the urinary cortisol levels following the symptom reduction. Results show a symptom reduction with a PTSD remission associated with higher urinary cortisol levels. These results confirm the EMDR treatment effectiveness with PTSD and the utility of including neuroendocrine measures in the assessment of treatment outcome in PTSD.

Keywords: Abuse Research

Accuracy Verified: Yes

12. Midboe, A., Benight, C., Harding, A., Iwaishi, S., & Johnson, L. (2002, November). A trauma-based treatment intervention for domestic violence victims. Poster presented at the 18th annual meeting of the International Society of Traumatic Stress Studies, Baltimore, MD.

Language: English

Format: Conference

Abstract:
Twenty-four participants from a local domestic violence center were selfselected into an individual therapy-plus-standard advocacy group (P/SA) or a standard advocacy (SA) group. The center’s advocacy staffed administered treatment for the SA group and the P/SA group. In addition to the SA treatment received by individuals in the P/SA group, there were also six licensed therapists who provided a manualized treatment incorporating CBT and EMDR. Symptoms of PTSD, depression, CSE, self-esteem, and coping skills were assessed.Additionally, saliva sample measurements were taken to examine the effect of treatment on the regulation of salivary cortisol. Results indicate that participants in the P/SA treatment condition experienced a significantly greater reduction in intrusive symptomatology and overall level of PTSD symptom severity, as well as a greater increase in CSE over time than those individuals in the SA condition. Moreover, changes in CSE were related to changes in psychological symptoms in individuals in the P/SA group from pretreatment to posttreatment and at follow-up.Almost no linear relationship between changes in CSE and other psychological variables in the SA group emerged.A minimal impact of treatment on coping skills was observed. There was a move towards a greater regulation of salivary cortisol at posttreatment in the P/SA group.

Keywords: Domestic Violence Poster

Accuracy Verified: Yes

13. van der Kolk, B. A., Hopper, J., Spinazzola, J., Blaustein, M., Hopper, E., & Simpson, W. (2003, October/November). Treatment outcome of fluoxetine vs. EMDR in PTSD. Symposium conducted (B. A. van der Kolk, Chair) at the 19th annual meeting of the International Society for Traumatic Stress Studies, Chicago, IL.

Language: English

Format: Conference

Abstract:
Treatment Outcome Studies of PTSD: This symposium presents three large carefully controlled treatment outcome studies using four different treatment modalities (CBT, EMDR, psychopharmacology and Cognitive Processing) and presents data on comparative efficacy, treatment responsiveness and resistance, effects on comorbidity, quality of life, and biological changes that accompany symptom improvement.

Treatment outcome of fluoxetine vs. EMDR in PTSD: This NIMH funded study compared the efficacy of two widely different treatment approaches for treating patients with PTSD: fluoxetine, which acts directly on biological systems (N=30), and Eye Movement Desensitization and Reprocessing (EMDR) (N=30). There also was a pill placebo control group (N=30). We assessed subjects with a multi-modal biological and psychological assessment, in order to determine whether treatment efficacy is associated with changes: 1) social adjustment, 2) psychophysiological reactivity to personalized trauma scripts (heart rate and skin conductance), and 3) basal salivary cortisol. We also tracked the stability of symptom change for nine months following the cessation of active treatment. Preliminary results suggest that at the end of 8 weeks of treatment, there is a 30% improvement in the pill placebo condition, while both active treatments demonstrate additional symptom improvement, with EMDR being most effective for the treatment of acute PTSD, and Prozac for subjects with prolonged childhood histories of trauma. Clinically significant improvement in CAPS scores is accompanied by an increase in basal cortisol and improvement in social and occupational functioning. We will also present data on the differential rates of symptom change in the different PTSD symptom clusters between the two treatment groups during the nine months of follow- up after cessation of the acute treatment phase.

Keywords: Fluoxetine Posttraumatic Stress Disorder PTSD Symposium

Accuracy Verified: Yes